Scientists Discover Why Most People
Are Genetically Predisposed To Obesity!

by Dr. Peter H Kay
(Preston, UK)

Worldwide, obesity has reached epidemic proportions. According to the World Health Organisation, more than 1 billion adults are overweight and at least 300 million of them are clinically obese.

Obesity poses a major health risk because people who are overweight are more likely to develop serious diseases such as heart disease, hypertension, stroke, type 2 diabetes and cancer. So what has caused this obesity epidemic?.

We all recognise a minority of people who can eat heartily, including "junk" foods and yet maintain a healthy body mass index (BMI). Why cannot we all be like those people? Scientists are beginning to understand why. It appears that evolutionary forces have genetically predisposed most of us to gain weight very efficiently.

So, why is this genetic predisposition to become overweight so common when a lower BMI is healthier? From an evolutionary perspective, it does not seem to make sense. To understand why, we must take a closer look at the interaction between the environment and our genetic blueprint throughout evolutionary history.

The story began to unravel about fifty years ago. At that time, a young researcher by the name of Winifred Doane was on a mission to find "famine-survival" genes. In Nigeria, she found some unusually fat flies. She discovered that the fat flies had a gene called adipose (ADP) that did not work so well.

The ADP gene has since been found in many different species. Reduced activity of the ADP gene leads to fat accumulation (see Ref. 1 and references therein]. In 2003, the ADP gene was discovered in humans 2, however it was not known whether the human ADP gene (now called WDTC1) was involved in any way in fat accumulation as had been found in other species.

Recently, scientists at Tufts University solved the mystery. They discovered that most of us have inherited a sluggish ADP gene that enables fat to be stored in our tissues very easily and slows down the way that fat it is burnt off or turned into energy 3. So, where did our sluggish ADP gene come from?.

It is thought that our poorly acting ADP gene originated millions of years ago and, over time, increased in frequency because it gave our ancestors an important survival advantage.

The sluggish gene enabled what little food was available to be readily stored as fat. Fat deposits could then be used as a source of energy and help survival in times of famine. Those with a more active ADP gene would be more likely to perish.

In modern times though, in areas of the world where, for the first time in human history, food is plentiful and refined, the sluggish ADP gene has become a health problem because it continues to store our food as fat, leading to obesity and development of diseases such as late onset diabetes

Simply making the ADP gene work better would be an effective way to lose weight, reduce fat deposition and maintain a healthy BMI and help to avoid development of diseases such as late onset diabetes.

Homeovitality Super Weight-Loss product, the health supplement developed in Australia by the world renowned scientist Dr. Peter H Kay has been formulated to do exactly that. It is based on sound scientific principles including the proven drug free safe gene targeting system of New Homeopathy, developed by Dr. B. Marichal. It is the only weight loss aid in the world that has been specifically designed to help our ancestral ADP gene to work better.

The Homeovitality Super Weight-Loss product has been scientifically formulated to help people lose weight more comfortably by also targeting the LEP gene. LEP reduces the feeling of hunger so it is easier to eat less food

The revolutionary Homeovitality weight loss product can be obtained via eBay and searching under "homeovitality" or online by contacting www.homeovitality.co.uk

More information can be found by accessing the www.wellnesshp.com website.

The author can be contacted on peterhkay@gmail.com.

References:

1. Suh J.M. et al., Adipose is a conserved dosage-sensitive antiobesity gene. Cell. Metab., 6:195-207, 2007.
2. Hader T, Muller S, Aguilera M, et al. Control of triglyceride storage by a WD40/TPR-domain protein. EMBO Rep., 4:511'516, 2003.
3. Lai C.Q et al., WDTC1, the ortholog of Drosophila adipose gene, associates with human obesity, modulated by MUFA intake. Obesity, 17: 593'600, 2009.